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Transdermal estradiol, delivered through patches or gels rather than oral tablets, bypasses the liver entirely, avoiding the first-pass hepatic metabolism that oral estrogen undergoes and that is associated with increased clotting factor production and thrombotic risk

As gut enterocytes represent a major source of DPP-4 within the gut mucosa, and the key intestinal cell type responsible for lipid uptake and TRL secretion, we studied lipid tolerance in Dpp4 Gut/ mice (20)

In vitro studies have shown that addition of DPP-4 is associated with impaired insulin signaling in primary human adipocytes, skeletal muscle, and smooth muscle cells (79), while conversely, silencing Dpp4 (using short interfering RNA) resulted in improved adipocyte insulin responsiveness (89)

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